A clotting cascade is formed when anticoagulants prevent the formation of blood clots in the body

A clotting cascade is formed when anticoagulants prevent the formation of blood clots in the body. The mechanism that follows after an injured blood vessel gets constricted is, right after the coagulation phase begins, which occurs because of the catalyzed enzymes. Plasma requires the fibrin thread to get formed, so the two major plasma proteins can help form a mesh-like framework for the clot. When the red blood cells get trapped while flowing towards the blood vessel, the clot is formed. Anticoagulants are divided into four different groups, such as Direct thrombin inhibitors, or Heparin. These two medications are used in case of deep vein thrombosis or to prevent red blood clots from forming during atrial fibrillation. The understanding of coagulation comes with secondary hemostasis which involves the Extrinsic Pathway, the Intrinsic Pathway, and the common pathways. The extrinsic pathway is activated by external trauma, which is more quicker in causing the blood cells to escape from the vascular system. Whereas, the Intrinsic path gets activated by platelets, that are noticed to be slower. The common pathway finishes the clot production when both of the other two pathways meet and finish.
Cardiovascular system works to control blood pressure so that the proper amount of oxygen and nutrients can reach different parts of our body. Blood pressure is adjusted through Cardiac output due to changes in heart rate or stroke volume. The cardiac output consists of regions which stimulates it through increasing contractility or inhabits it for a decreased heart rate. The Peripheral resistance, on the other hand, is described as the resistance of the arteries to blood flow. The strength of the arteries is dependant on the increasing and decreasing constriction. These are determined by autonomic activity, pharmacologic agents, and blood viscosity.